S.A.L.T. Device




Supraglottic Airway Laryngopharyngeal Tube



Link to product: S.A.L.T.


Some videos:







I have used the S.A.L.T. device once on a cardiac arrest patient. Initially it found it's way in the right mainstem bronchus; which we easily resolved. Others have told me that they have had problems with the securing device. Some have stated that they have had trouble avoiding esophageal placement. I am not certain if the S.A.L.T. device will replace the King LT as my choice for cardiac arrest victims, but it is an interesting product nonetheless. It may have it's place in primary respiratory arrest. I am still a fan of videolaryngoscopy at the moment, even if it is the most expensive option.



66 year old male CC: Chest pain

Here's another great case submitted by Nick Ciaravella of Grady EMS in Atlanta, GA.

66 year old male presents to EMS with chest pain.

S - Chest Pain
A - None
M - Atenolol, HCTZ
P - HTN
L - meal, 7 hours prior to event
E - Mowing his lawn

O - Started while mowing his lawn
P - Provoked while exerting himself, Palliated initially when he sat down to rest
Q - Sharp
R - Substernal, initially radiating to his jaw, when he rested the pain was only in his chest
S - Initially 10/10, upon ems arrival 4/10, en route 8/10, 9/10, and 10/10 upon arrival at ED
T - No previous episodes

The patient initially presented to EMS with 4/10 pain and vitals as follows, 148/84, pulse 72, 18 respirations, SPO2 96%, Lung sounds clear and equal, BGL 103.

The patient was placed on 3 LPM O2 via NC, given 324 mg Aspirin PO, given 0.4 mg Nitro Tablet Sublingual and then 1 inch of Nitro Paste Transdermal. The Patients pain increased en route to the ED and began to radiate down his left arm en route.

12-lead ECG #1


12-lead ECG #2 (about 15 minutes later)


What do you think?

See also:

Anterior ischemia or posterior STEMI?

26 year old male CC: Chest pain

74 year old male CC: Chest pain

50 year old male CC: Respiratory distress, chest pain

48 year old male CC: Chest discomfort, shortness of breath

Pure (Isolated) Posterior STEMI -- not so rare, but often ignored! - Dr. Smith's ECG Blog

Empathetic vs. Pathetic


Empathetic vs. Pathetic
Listen up EMS

By Adam Thompson, EMT-P



I know I have pretty much kept this blog purely aimed towards education, fact, and evidence. It is time for a rant though.

Please read the following links before continuing:
So what do we do? How do we change the attitude of our fellow EMSers? Do we need to make the change, or do they?

This is something I have been cognizant of for some time. Being an overachiever in EMS gains you no friends. The more successful you are, the bigger your blip is on the radar of ridicule. But who cares, right? They aren't talking about you for being a cretin medic that screws up on calls.

Example. I am a young, but experienced medic. I have achieved a lot in my career. I am a published author. My training captain recently sent out one of my articles with a thumbs up message to my entire agency. The response was as good as it was bad. The ongoing joke is with every conversation I bring up I hear "why don't you go write an article about it". Some of this may be just a joke, but I can feel the animosity from many. Why?


The Problem

I think, from my experience I have pinned down one problem. We are our own bosses.

I know we all have bosses, chiefs, supervisors, what have you. What I mean is, most of us don't have those people on our trucks with us. If you work in a system like mine, you may be the lone medic working with an EMT, or maybe you are the EMT. I think that the systems that have multiple medics per ambulance suffer less from these issues--and here's why.

If you are use to making your own decisions with little repercussion and the ignorant feeling of correct-fulness, you will not likely be inclined to take advice from your fellow medics. I dread the response of a peer that I attempt to assist with a smidgen of education. Because there is a naive belief that they know EVERYTHING.


Why are we so damn sensitive?

If you haven't read my letter to the new guy, go read it. It is time we toughen up. If a salesman isn't making a company any money, are the bosses going to be fearful to approach him?

I was speaking with one of the white shirts (officer) from the training department the other day and made some proposals. I said we should have a real QI/QA committee that picks ten calls at random every month. Some ran good, some not so good. Then, the medics on each of those calls would have to present each case in front of their peers. There would be questions and answers.

My thought was that we hear about the bad calls through hearsay all the time, but do those medics get to defend themselves on a normal basis--no! A lot gets lost in translation. Sometimes you have to be on a call to understand, right? Well here is the chance to remedy that while implementing a QI/QA process that physicians use and grow from.

His response: The union will never allow it.

What the hell are we doing to ourselves? While unions might be established to protect the best employees, why do they work so hard to keep the worst? We can learn so much from each other, but you can't learn if you keep thinking there is nothing left to learn.


We all mess up. Get over it!

I consider myself a pretty educated paramedic. I have made many mistakes.

Now think about that. When do you learn most? I'm not saying that there is a cemetery somewhere, filled with all my mess-ups. I'm talking about simple, little mistakes. Mistakes that if unmade, would have lead to more information and a faster diagnosis or better treatment modality.

If you think you are invincible, go ahead and continue living on your beachfront desert property. You make mistakes too.

If you can learn so much from your mistakes, and I can learn so much from mine, why can't we BOTH learn from EACH OTHER'S mistakes? This of coarse requires a deflation of bulbous craniums.


When did this stop being about the patients?

Empathy is a virtue that is quickly finding itself on the endangered attributes list.

Please read Professionalism: What we say by me.

No matter what you read here, or believe. No matter how long you have been doing this. No matter how bitter you are. You have to agree that at some point of your career you wanted to help people. You wanted to make a difference, and do some good. So I ask you this... Are you?

PowerPoint Presentation on Strain Patterns!

This is an awesome PowerPoint presentation from one of my favorite ECG textbooks, 12-Lead ECG - The Art of Interpretation.

You owe it to yourself to purchase this book (and no, they don't pay me to say that).

Make sure you use "full screen" so you can see all of the features of this important presentation. Pay special attention to slides 33-47!

Slide 45 shows a strain pattern from left ventricular hypertrophy (LVH).

Slide 47 shows a split screen with a strain pattern from right ventricular hypertrophy (RVH) on the left and acute infero-posterior STEMI on the right.

This is the book that taught me how to recognize strain patterns! So pay attention because this is one of the most important STEMI mimics!




See also:

Left ventricular hypertrophy - Part I

Left ventricular hypertrophy - Part II

Right ventricular hypertrophy vs. isolated posterior STEMI

Comment on Intravenous morphine at 0.1 mg/kg is not effective for controlling severe acute pain in the majority of patients





In response to Intravenous morphine at 0.1 mg/kg is not effective for controlling severe acute pain in the majority of patients, there is a comment by medic.


thank you for your post. i'd like to add a few thoughts, and please feel free to comment on them.



Thank you.


1. i have a suspicion that pts who rate their pain 7-8/10 tend to be more honest about their pain than the ones who rate their pain 10/10 (worst pain ever), which perhaps partially explains the study's findings when sorted by initial pain rating.



That may be. Pain is subjective. Out of 119 patients, only 5 rated their pain 7 out of 10, so this is a small fraction that may not indicate anything. If we wish to draw conclusions about patients with 7 out of 10 pain, we need to set up a much larger study and propose our hypotheses before the study is begun.

Even if a report of 7 out of 10 pain is more honest than a report of 10 out of 10 pain, does that make it any less appropriate to treat 10 out of 10 pain aggressively?


2. competence is a huge factor is any setting, not just ems. there are plenty of (supposedly better-trained) docs and nurses who are clearly retarded.



Retarded is not the right word. However many doctors, nurses, and medics just do not get it. Maybe pain management has not been explained to them in the right way.

Many doctors do seem to become much more comfortable using opioids to treat pain after experiencing severe pain themselves.

We never seem to hear about doctors becoming less comfortable using opioids after experiencing severe pain.

This suggests that there is something important that is not understood by the doctors until after experiencing severe pain.

I suspect that studying this might require a huge sample of doctors, just to be able to track the change in prescribing/ordering habits vs. personal experience of severe pain, whether their own pain or the pain of someone they care deeply about.


3. i personally have a high threshold for when i break the narcs open, as i too work in a poor area. that's not to say poor people can't have pain; that's just taking into account other factors such as a seemingly higher rate of drug use/abuse.



It isn't our pain threshold that matters, but the patients' pain threshold.

Opioids are not the appropriate treatment for all pain, but it is appropriate to treat severe pain aggressively with opioids when nothing else works (such as when nothing else is available). Nitrous oxide is something that can be safely used that might significantly decrease the amount of morphine needed to manage severe pain.


4. once i make the decision to use narcs, i am not stingy with them as experience shows that prehospital morphine doses are clearly inadequate. the more important issue here is a training crews for a heightened awareness of the potential for respiratory depression and allergic reactions (just had one last week).



And what did you need to do for the allergic reaction? Diphenhydramine?

Just because something happened last week, does not mean that it is common. We need to be aware of the potential for allergic reactions with all of our medications.

One of the many advantages of fentanyl, over morphine, is that fentanyl is much less likely to produce an allergic reaction.

Recognizing and dealing with respiratory depression should not require anything more than competently monitoring the patient.

Many, but not all, patients will experience respiratory depression with appropriate pain management, because pain tends to stimulate a sympathetic response. Having respirations decrease to normal is a good thing, even though this is respiratory depression.

If the patient shows signs of inadequate oxygenation/ventillation due to respiratory depression, then all that needs to be done is to get the patient to talk. Find a subject that the patient is interested in, people love to talk about themselves, and get them to keep talking. Or just keep asking questions that are not answered with a nod, or shake, of the head. Ask orientation questions. Even just telling the patient to take a deep breath every so often will work.


A talking patient is a breathing patient.


5. it's a big training issue to get crews to recognize those pts who are in pain and those who are trying to score narcs. this is where experience counts and it's difficult to teach. that being said, it's risky to presume that people are trying to score narcs.



We should not presume that people are trying to manipulate us to give them drugs inappropriately. We should be aggressively looking for indications that the patient has legitimate pain.

If I need to give out morphine and fentanyl to a bunch of junkies in order to avoid missing some patients with legitimate pain, then I will be the candy man.

Let me put this in perspective.

If I need to give out albuterol nebulizer treatments to a bunch of people who do not need nebulizer treatments in order to avoid missing some patients with legitimate asthma/emphysema/bronchitis, then I will be the nebulizer man.

If I need to give out IV dextrose to a bunch of people who do not need to receive dextrose through an IV in order to avoid missing some patients with legitimate hypoglycemia and an inability to take glucose by mouth, then I will be the dextrose infusion man.

If I need to take some people with minor injuries to a trauma center in order to avoid missing some critical trauma patients, then I will be the minor trauma man.

I am not encouraging over-treatment, for the sake of over-treatment. We do need to be much better at assessment, rather than treating mechanism. How much training do we have at recognizing drug seekers, who are not seeking drugs for legitimate pain? If we are trained at this in paramedic school, or on the job, what are the qualifications of the person providing this training? What research has been done to demonstrate the accuracy of the methods of differentiating between legitimate drug seekers and illegitimate drug seekers?

The best way to make someone a drug seeker may be to under-treat their pain.

Our concern needs to be much less on being police and much more on being paramedics.


.

62 year old male CC: Chest pain

62 year old male presents to the emergency department complaining of chest discomfort.


Past medical history is significant for dyslipidemia and ulcerative colitis. Also prior history of significant tobacco use. Maternal history of CAD. Maternal and paternal history of CVA.

The patient's only medication is Lipitor but he took an aspinin en route to the hospital.

Onset: Patient states the pain started that morning and became progressively worse since lunch time.
Provoke: Nothing makes the pain better or worse.
Quality: Sharp and nonpleuritic
Radiate: The pain radiates down the right arm to the bicep.
Severity: 7/10
Time: Patient states he experienced a similar pain in his right upper chest several days prior while playing tennis. He stopped exercising and the pain resolved.

The pain makes the patient feel "a little clammy." He denies shortness of breath. He states that he feels "a little dizzy" but denies palpitations. He had a negative stress test 3-4 years ago.

He has a known history of left bundle branch block.

The patient's skin is warm and dry.

Breath sounds clear bilaterally. No JVD.

Neuro exam normal.

Vital signs:

Resp: 18
Pulse: 60
BP: 140/72
SpO2: 98 on RA

A 12-lead ECG is captured and presented to the ED physician within 5 minutes of arrival.



An "old" ECG is pulled from the computer system for comparison.


What is your impression?


** Update 05/19/2010 **

After oxygen and nitroglycerin the patient reports a significant decrease in pain.

An additional 12-lead ECG is captured.


There is now slightly less ST-elevation in leads V3 and V4.

Remember that a secondary ST-segment abnormality (as opposed to a primary ST-segment abnormality) should not "improve" with oxygen and nitroglycerin!

In other words, if this ST-elevation was caused just by the LBBB, it shouldn't be "getting better". Changing ST-segments suggest the dynamic supply vs. demand characteristics of ACS!

Now, let's go back to the initial 12-lead ECG. Is the ST-elevation in the anterior leads cause for concern?

Go back and read Identifying AMI in the presence of left bundle branch block (or paced rhythm). Remember, discordant ST-elevation = or > 5 mm is the least specific of Sgarbossa's criteria! That's why we use the modified rule that I learned from Dr. Stephen Smith of Dr. Smith's ECG Blog.

That criterion states that discordant ST-elevation should not be more than 0.2 (or 20%) the depth of the S-wave in the setting of left bundle branch block (ST/S ratio).

Using that criterion, how does this ECG measure up? Let's take a look.


Ladies and gentlemen, we have a winner!

The patient was ultimately cathed and angiography revealed 100% occlusion of the LAD.

Final thought:

Does it get any more difficult that that? If Dr. Smith's decision rule works this great, shouldn't we be shouting it from the rooftops?

See also:

Discordant ST-elevation in LBBB or paced rhythm

Identifying AMI in the presence of LBBB

Sgarbossa's criteria - new graph

"New" LBBB - What's the big deal?

New left bundle branch block is a poor indicator of coronary occlusion - Dr. Smith's ECG Blog

STEMI best seen in PVC (Dr. Smith's ECG Blog)

Prehospital use of analgesia for suspected extremity fractures




This is an older study that puts the prehospital pain management problem into a bit of perspective. While prehospital pain management has improved a lot in some places, other places may still be handling pain as described in this study. This is only ten years old. Attitudes are not changed so easily.

The authors looked at what is probably the least controversial type of pain management. If you were to ask medical directors what they feel most comfortable having paramedics use opioids to treat, the only other choice is likelty to be pain due to burns. Chest pain became a bit controversial after the CRUSADE study, but I will get to that in another post.


Over the last decade, pain and its management have received considerable attention. Most notably members of the medical profession in general and specifically emergency medical professionals undertreat pain to a considerable extent.3[1]



While I would love to be able to defend everyone from this charge euphemistically referred to as undertreatment, the reality is that a decade later, the problem has not changed that much.


This was an observational study involving a retrospective review of all emergency medical services (EMS) runs for suspected extremity fractures[1]


During the study period, all EMS run reports were evaluated by the fire department’s quality assurance coordinator. Only reports documenting the paramedic’s impression that the patient had sustained a fracture, or suspected fracture, of any extremity were included in the database.[1]



There is no mention of any requirement to document any kind of measurement of pain. This seems to be the most significant problem with pain management in the system studied. How do we assess the quality of pain management if we do not assess pain?

The whole structure of this study revolves around the apparent inability to assess pain. There are a bunch of conclusions drawn. Here is what may be the most important omission of the study.

If we do not assess something, how do we treat it appropriately?


If a medical director does not make it clear that pain assessment and management are taken seriously, then is there much reason to expect the paramedics to be more aggressive than the medical director?

We need to provoke medical directors, emergency physicians, emergency nurses, EMTs, and paramedics to take pain seriously.

It's not my pain.


Akron Fire EMS employs a two-tiered transport system whereby nonurgent patients who may be safely transported in a private vehicle are deemed code 1, allowing the med unit to be put back into service. Nonurgent patients who require ambulance transport to the hospital become code 2 and their care and transport to the hospital are transferred to a private ambulance. A patient requiring immediate transport, medication, or procedures rendered by a paramedic is transported directly to the hospital as a code 3.[1]


The two-tiered structure of this EMS system may unwittingly serve to negatively affect the administration of pain medication in several ways. Administering pain medication to a code 2 patient, for whom transport to the emergency department would be transferred to a private ambulance, would automatically change the run to a code 3. Upgrade to a code 3 would necessitate transport directly to the emergency department by the treating paramedic squad, extending the time required to complete the run.[1]



Another question is whether code 3 means a lights and sirens transport to the ED. What extremity fractures, other than those cutting off circulation, require such rapid and rough transport? Even fractures cutting off circulation are unlikely to benefit from the slight difference in transport time that lights and sirens would provide. Slightly faster, but much rougher and much more painful in spite of the pain medicine! Why?


How did this service do at using pain medicine to manage suspected extremity fractures, which are expected to have a high correlation with severe pain?


A total of 18 patients (1.8%) received treatment for pain; nitrous oxide was administered to 16 patients (1.6%), and morphine sulfate to two patients (0.2%).[1]



We know that 16 patients (out of a thousand patients with suspected extremity fractures) received nitrous oxide and 2 patients (out of a thousand patients with suspected extremity fractures) received morphine.

We do not know if they were being treated for pain, since there is no indication of any assessment of pain. We expect that the patients with suspected extremity fractures would have a lot of pain. If you have ever had a painful extremity fracture, you might expect all extremity fractures to be painful. You might also desire that many, most, or even all of these suspected extremity fractures be treated with pain medicine. The authors do not provide anything to support this conclusion.

Let's look at the injuries documented.




All of them seem as if they would be painful fractures. Still, we do not know anything about the pain of these patients.

What else was done that might have acted as pain management?


Supportive medical treatment provided included air splints (25% of patients); full immobilization (19%); ice packs (17%); bandages/dressings (16%); and intravenous lines (9.4%).[1]



Air splints may help to relieve pain by positioning the extremity in a less painful, assuming there is any pain, position. Splinting may temporarily increase pain during application.

Full immobilization is unlikely to provide any pain relief. Full immobilization on a solid long spine board is expected to increase pain.[2]

Ice packs can increase pain, decrease pain, or both.

Bandages/dressings might provide some stabilization, or pressure, that decreases the sensation of pain.

Intravenous lines are often painful. If I only suspect an extremity fracture, I would not have any other justification to be starting an IV, except to have a route to deliver IV pain medication. 2 patients received IV pain medication. 94 patients received IV lines. 2/94?

Did one medic start a line, while the other medic contacted medical command. Since The care of each patient is discussed with an online medical control emergency physician in a local emergency department, the superfluous medic may use that time to get online medical control emergency physician contact out of the way, so that the medic can do something useful, such as assessing or treating the patient.

Did the online medical control emergency physician give this counter-order to the medics? Do not follow your standing orders for pain management. Do not give any pain medicine.

Were the medics, or was one of the medics, hesitant to provide any pain medicine without first contacting the online medical control emergency physician?

Are the medics routinely yelled at by emergency physicians if they administer pain medicine without requesting permission first, even though protocols allow them to give pain medicine without asking for permission?

Do administrators receive complaints from emergency physicians when medics follow standing orders for pain medicine?

The number of patients receiving morphine is so small, that I want to know what was so bizarre about these patients that these Just say No! paramedics gave morphine.

Was the morphine given on standing orders?

Was the morphine even given intentionally?

Since giving morphine for suspected extremity fractures is such a freak occurrence in this system, is there any evidence to suggest that these were not 2 medication errors?

Is the occurrence of morphine administration any less rare than the system's occurrence of medication errors?

The use of morphine is so breathtakingly out of the ordinary in this system, that I do not see any reason to conclude that there is any connection between morphine and suspected extremity fracture. Were any other medications, aside from nitrous oxide, given to any of these patients? Were any of those medications given more frequently than morphine? 2/1,000 suspected extremity fractures.


The EMS pain control policy included standing orders for administration of either morphine sulfate (adult dose: 2–5 mg intravenous push [IVP], may repeat x 1; pediatric dose: 0.1 mg/kg) or nitrous oxide (50%), self-administered. The care of each patient is discussed with an online medical control emergency physician in a local emergency department. By protocol, analgesic therapy is contraindicated in patients with the following conditions: altered level of consciousness; alcohol or drug use; allergies to morphine or nitrous oxide; hypotension; head injury; chest injury with suspected pneumothorax; abdominal pain with possible bowel obstruction; symptomatic asthma or chronic obstructive pulmonary disease (COPD); or respiratory distress.[1]



Even with standing orders, patient care must be discussed with a doctor. In that case, are they really standing orders? There are a lot of contraindications. I almost expect to see suspected extremity fracture listed as a contraindication for morphine. How much different would the results be, if that were the case?

2/1,000 vs 0/1,000.

Is this number, 2/1,000, even close to being statistically significant?

We don't know how many of the 1,000 patients actually had pain that would be appropriate to treat with morphine.


This study examined the use of analgesia in 1,000 prehospital patients with suspected fractures of the extremities who were treated by paramedics. Of the 1,000 patients, only a very few (1.8%) received any pain medication, although morphine sulfate and nitrous oxide were available to the paramedics by both standing order and direct physician order through online medical control.[1]



I think it is misleading to suggest that there was any encouragement by medical command to treat patients with morphine. However, I have no way of knowing if one, or both, of the patients treated with morphine only received morphine because the doctor ordered it.

My experience with online medical command has been one of repeated refusal to give orders for for pain medicine for patients with pain - pain that I would be authorized to treat on standing orders under my current protocols.

What is the difference?

The patients treated with morphine do not suffer as much. The medical command physician does not get to exercise a medical whim to deny pain medicine purely due to the physician's lack of understanding of pain management.


The mean time spent on the scene for all patients in the study was 23 ±3.4 minutes. Scene times were significantly longer for patients who received pain medication (n = 18) 32.8 ±17.4 minutes, than for those who did not, 22.8 ±10.4 minutes (95% CI 5.22 to 14.58). Transport times to the area hospitals average 7 minutes in this system, with the three main receiving hospitals located in the same geographic area. [1]



Unless a patient is unstable (or at risk of injury if not moved), there is no good reason to transport the patient until after the pain is managed. It does not matter if this means an extra 5 minutes on scene or an extra hour on scene. More aggressive dosing (morphine 0.1 mg/kg followed by 0.05 mg/kg every 5 minutes until significant relief) and more appropriate medication (fentanyl at appropriate doses) will result in less time on scene. We should not be manipulating painful injuries until after the pain is managed, unless there is some good reason. That is rare.

When I call for orders for more pain medicine, because the standing orders have not been appropriate in managing pain, medical command often wants to know how far I am from the hospital. My response is, That depends on how quickly I get orders for appropriate pain management, because the patient is not being moved until the pain is managed. Why isn't that obvious to everyone?

Why increase the patient's pain to move the patient to bring the patient to the pain medicine in the hospital, when the patient can be treated just as safely, if not more safely, before being moved?


Second, the administration of nitrous oxide requires that in addition to directly transporting the patient, the paramedics must also exchange the used nitrous tank for a new one. In Akron, the only tank exchange site was located in a remote part of the city, necessitating extended duties and travel time for one of the paramedics. Upon completion of this study, replacement nitrous tanks were placed in each of the 12 fire houses to facilitate more convenient restocking.[1]



It is good to see that they are trying to make things better for patients by eliminating the excuses used by paramedics, when medics rationalize avoiding treating patients appropriately.


Managing pain in the prehospital setting may require a multifaceted approach. Pain experienced by the patient must be evaluated in an objective manner, and once assessed, managed appropriately. Prehospital care providers should be encouraged to appreciate their patients’ pain and given the tools and affirmation needed to provide the most appropriate care.[1]



Yes.


CONCLUSION
Prehospital care providers and their medical control supervisors have room to improve the quality of pain control in the prehospital setting. In this review of the use of analgesia for patients with suspected fractures of the extremities, pain medication was rarely used. Improvements in both the recognition and assessment of pain and in treating the pain in the prehospital setting are slow to be implemented. Education, pain control evaluation, protocol development, and quality assurance and audit systems are all measures that can be used to improve the quality of pain management in the prehospital setting.[1]



All good points, but the most important point is not in there.


Pain management is about treating pain, not treating specific medical conditions.


If you look at all of the contraindications to the use of pain management in this study, there appears to be a strong bias against treating many painful conditions that are not medical contraindications. These appear to be just demonstrations of discomfort with pain management and ignorance of appropriate pain management. As critical as I am of this study, at least the authors are working to improve the way their system manages pain. Most systems seem to deny that there is a problem.

We need to educate prehospital providers to be much more aggressive with pain management.

We spend so much time worrying about paramedics being too aggressive with pain management, but nobody seems to be able to come up with any evidence to support this paranoid fantasy.

We need to provoke medical directors, emergency physicians, emergency nurses, EMTs, and paramedics to take pain seriously.


Footnotes:


^ 1 Prehospital use of analgesia for suspected extremity fractures.
White LJ, Cooper JD, Chambers RM, Gradisek RE.
Prehosp Emerg Care. 2000 Jul-Sep;4(3):205-8.
PMID: 10895913 [PubMed - indexed for MEDLINE]


^ 2 Unnecessary out-of-hospital use of full spinal immobilization.
McHugh TP, Taylor JP.
Acad Emerg Med. 1998 Mar;5(3):278-80. No abstract available.
PMID: 9523943 [PubMed - indexed for MEDLINE]

Standard backboard immobilization is not harmless and can cause significant pain, especially at the occipital prominence and lumbosacral areas. Within 10 minutes of being placed in FSI, Hamilton and Pons12 showed that volunteers developed moderate to severe pain. After 30 minutes in FSI, Chan et al.13 found 100% of volunteers complained of pain, with 55% of the group grading their pain as moderate to severe in quality. Interestingly, 29% of the subjects developed new symptoms over the course of the next 2 days. Chen et al. concluded that “the standard process of immobilization may complicate the evaluation of the trauma patient by generating additional symptoms . . . leading to unnecessary laboratory tests and radiographic studies, time of immobilization, and ultimately, health care costs.” In addition to pain, FSI can cause changes in pulmonary function. can cause pressure ulcers of the buttocks, scalp, or neck, and can increase the risk of aspiration after vomiting.13,14 Because standard FSI can compromise maternal and fetal circulation, it is relatively contraindicated in gravid women.

^ 12 The efficacy and comfort of full-body vacuum splints for cervical-spine immobilization.
Hamilton RS, Pons PT.
J Emerg Med. 1996 Sep-Oct;14(5):553-9.
PMID: 8933314 [PubMed - indexed for MEDLINE]


^ 13 The effect of spinal immobilization on healthy volunteers.
Chan D, Goldberg R, Tascone A, Harmon S, Chan L.
Ann Emerg Med. 1994 Jan;23(1):48-51.
PMID: 8273958 [PubMed - indexed for MEDLINE]


^ 14 A review of spinal immobilization techniques.
De Lorenzo RA.
J Emerg Med. 1996 Sep-Oct;14(5):603-13. Review.
PMID: 8933323 [PubMed - indexed for MEDLINE]



.

EMS Week 2010



Happy EMS Week everyone!!!

In the comments, please provide your most memorable experience from this last year of being an EMSer.


The following video was something provided by (the old) Rocky Mountain Medic. He was a fellow EMS blogger, and wanted this shared for EMS Week.

67 y/o male CC: Syncope



Also posted over at 12-Lead ECG Blog, go check out all the other great stuff there!


A 67 y/o male has fallen to the ground at his residence. His "partner" called 911 after seeing that he was unconscious. Upon your arrival the patient is alert and requesting that you pick him up because he really needs to make a bowel movement. The patient denies syncope but states that he does not remember falling.

Here is the initial ECG and the subsequent 12-lead ECG. Sorry for the poor quality.





What do you see?

What do you want to know?

What do you want to do?



****Update****



His Vital Signs

HR correlates with monitor, pulse not palpable at radial.

Initial BP 78/60

AAOx3, normal mental status, just wants to make a bowel movement.

Skin - Pale, more pronounced and white from the waste down. Skin was relatively dry.




****Update 5/20/2010****


A new 12-lead ECG is captured during transport.

The patient's vital signs do not improve dramatically with IV fluids.




****Update 5/23/2010****

During transport the patient's condition declined rapidly. After the 12-lead ECG above was captured, the patient went in to a decorticate posture. As most of you know, this is indicative of some sort of neuro compromise. With his airway control, mental status, and respiratory rate all declining, brainstem herniation was at the top of the list of differentials.

The patient became pulseless and apneic just prior to arriving at the ER--according to my partners, just after he released the bowel movement. The patient was not revived.

So what happened here?

Me and the field training officer came up with a few possible solutions. First, there is ST-elevation in the inferolateral leads of the initial 12-lead ECG. With the hypotension, a RCA occlusion is a possibility. If the patient has a dominant RCA, there appears to be some ST-depression in the septal leads, but this is a RBBB pattern, so with the T-wave discordance, the ST-depression is not a good clinical indicator of posterior wall involvement.

First Possibility: Right-sided infarct with hemodynamic compromise leading to a syncopal episode. The syncope caused a secondary head injury which cerebrally herniated during transport. I would like to note that this is highly unlikely. Also, the patient did not improve with fluids, which would have happened with a traditional RV infarct.

Second Possibility: It is much more likely that the patient had an atypical hemorrhagic stroke that presented with the first symptom of syncope. The changes on the 12-lead ECG could just be concurrent with cerebral ischemia. This is not completely understood, but theories involving nerve endings in the myocardium are abundant. The patient's ICP would have increased during transport with the final result being cardiac arrest.

Third Possibility: Abdominal aortic aneurism with severe secondary cerebral ischemia due to hemodynamic instability. I'm not fond of this idea even though the AAA fit the picture in the beginning, it does not explain the decorticate posturing.


We also keep the huge possibility that we have no idea what happened on the list. Ok, so I wish I had more to give you, but an autopsy was not performed on this patient. It remains a mystery.

Improving AHA



Those of you who follow the Paramedicine 101 Facebook fan page may have noticed a discussion, after this post was shared on the wall. A follower mentioned the AHA Learn: Rapid STEMI ID course, and how it could be a solution. Myself, and Tom B then casually shared some choice opinions about video-based AHA courses. I recently received an email from the commenter, who happens to be affiliated with the AHA. He humbly asked if he could call me regarding my ideas. I suggested a conference call with Tom, and he suggested a conference call with the people whom make decisions at AHA. If we do get them to take interest, I would like to be able to provide a lot of insight. I have many ideas, but would like to solicit some more from our faithful readers.

How do you feel AHA courses could improve?

- ACLS
- CPR
- PALS
- Rapid STEMI ID

Please provide any suggestions. This is our chance to make a difference. RM, don't hold back.